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Can Someone Explain To Me What "delayed cord cutting" Does?

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Asked by PrncssTmmy at 9:07 PM on Jul. 11, 2011 in Pregnancy

Level 4 (52 Credits)
Answers (14)
  • i have read that itll give the baby a chance to get more blood intheir system before you cut so that they are less likely to be anemic trying it out this pregnancy to see if it helps

    Answer by bamamommy2009 at 9:11 PM on Jul. 11, 2011

  • it's so all of the blood from the cord can go into the baby, they mostly will do this for Preemies. The only risk is the risk of too much blood entering the baby.

    Answer by DomoniqueWS at 9:19 PM on Jul. 11, 2011

  • There are literally dozens of studies which show NO increase in anemia, in fact some show a decrease, of jaundice. And there are a few which show an increase in the need for phototherapy

    Yes there are studies which show an increase in polycythemia, but not pathological polycythemia. Which suggests that there is just a lack of understanding of what is normal.
    Late vs Early Clamping of the Umbilical Cord in Full-term Neonates
    Systematic Review and Meta-analysis of Controlled Trials

    Eileen K. Hutton, PhD; Eman S. Hassan, MBBCh

    “Conclusions Delaying clamping of the umbilical cord in full-term neonates for a minimum of 2 minutes following birth is beneficial to the newborn, extending into infancy. Although there was an increase in polycythemia among infants in whom cord clamping was delayed, this condition appeared to be benign.”


    Answer by Anonymous at 1:07 AM on Jul. 12, 2011

    the early effects of delayed cord clamping in term
    infants born to libyan mothers

    Musbah Omar Emhamed1, Patrick van Rheenen2, Bernard J Brabin1,2

    “No significant differences were found inclinical jaundice or plethora. Surprisingly, blood analysis showed that two babiesin the early clamping group had total serumbilirubin levels (>15mg/dL) that ne-cessitated phototherapy. There were no babies in the late clamping group whorequired phototherapy. Three infants in the delayed clamping group had polycy-thaemia without symptoms, for which no partial exchange transfusion was neces-sary. Delaying cord clamping until the pulsations stop increases the red cell massin term infants. It is a safe, simple and low cost delivery procedure that should beincorporated in integrated programmes aimed at reducing iron deficiency anae-mia in infants in developing countries.”


    Answer by Anonymous at 1:07 AM on Jul. 12, 2011

    The Effect of Timing of Cord Clamping on Neonatal Venous Hematocrit Values and Clinical Outcome at Term: A Randomized, Controlled Trial
    José M. Ceriani Cernadas, MDa, Guillermo Carroli, MDb, Liliana Pellegrini, MDc, Lucas Otaño, MDd, Marina Ferreira, MDa, Carolina Ricci, MDa, Ofelia Casas, MDc, Daniel Giordanob, Jaime Lardizábal, MDb

    “layed cord clamping at birth increases neonatal mean venous hematocrit within a physiologic range. Neither significant differences nor harmful effects were observed among groups. Furthermore, this intervention seems to reduce the rate of neonatal anemia. This practice has been shown to be safe and should be implemented to increase neonatal iron storage at birth.”


    Answer by Anonymous at 1:08 AM on Jul. 12, 2011

    Neonatal plethora, polycythemia, and hyperviscosity

    In a review of the literature, I found three gravity-drained cases of plethora clamped at 1 to 5 minutes that were cured by phlebotomy.” Uterine contraction was not documented. It is unclear whether the infants’ excess blood would have flowed back into the cord during uterine diastole if the clamp had been quickly removed.3
    Polycythemia (hematocrit >65%) is often produced by late clamping. Hemoconcentration normally follows placental transfusion. It also occurs during normal labor. Serum albumin and colloid osmotic pressure (COP) rise with the hematocrit. Pulmonary function requires a COP high enough to prevent pulmonary edema. At elective cesarean section with rapid cord clamping, this COP increase does not occur. Not surprisingly, wet lungs occasionally result.


    Answer by Anonymous at 1:09 AM on Jul. 12, 2011

  • Oligohydramnios results in vein compression, causing increased capillary pressure in the placenta, which leads to fluid loss, dehydration, and hemoconcentration. Amnioinfusion may correct this; otherwise, rapid fluid replacement at birth is needed to amend this pathologic polycythemia.

    The multifaceted (and uncertain) hyperviscosity syndrome11-13 is based on the premise that increased viscosity (high hematocrit) results in decreased tissue perfusion.2,11 However, factors other than viscosity also affect tissue perfusion.


    Answer by Anonymous at 1:10 AM on Jul. 12, 2011

  • According to Poiseuille’s law, the blood flow through vessels (liquid flow through tubes) is inversely proportional to the length of the tube and the viscosity of the liquid, and directly proportional to the pressure differential and to the fourth power of the radius of the tube. Therefore, if the radius is reduced from 3 to 2, flow is reduced 81:16, or by four-fifths; whereas, if viscosity is increased from 2 to 3, flow is reduced 1/2:1/3, or by one-sixth. Clearly, vasoconstriction reduces blood flow much more than a similar change in viscosity.

    In clinical practice, late clamping produces a high hematocrit, 2, 9,14,15 high blood pressure, and vasodilatation to accommodate the large volume of blood.9,16 These latter two factors should increase tissue perfusion. In searching the literature, I was unable to find any documented case of hyperviscosity syndrome in which the cord was clamped late,”

    Answer by Anonymous at 1:11 AM on Jul. 12, 2011

  • although I did find many documented cases of late clamping involving normal newborns with high hematocrits. 2,9,15,17,18

    There are, however, many documented cases of hyperviscosity syndrome with high hematocrits (e.g., cases involving gestational diabetes or postmaturity) in which the cord was clamped before physiologic cord closure, thus creating low blood volume, low blood pressure,16 and vasoconstriction coupled with the polycythemia.11 The inadequate tissue perfusion is blamed on the high hematocrit, when the root cause of the hyperviscosity syndrome is hypovolemic vasoconstriction enforced to the fourth power.

    1. American College of Obstetricians and Gynecologists. Umbilical Artery Blood Acid-Base Analysis. Washington, D.C.: ACOG; 1995. Educational bulletin 216.

    2. Linderkamp O. Placental transfusion: determinants and effects. Clinics in Perinatology 1982;9:559-592.


    Answer by Anonymous at 1:12 AM on Jul. 12, 2011

  • 3. Gunther M. The transfer of blood between baby and placenta in the minutes after birth, Lancer 1957;i:1277-1280.

    4. Yao AC, Lind J. Effect of gravity on placental transfusion. Lancet 1969;ii:505-508.

    5. Botha MC. The management of the umbilical cord in labour. S.A. J Obstet Gynecol 1968,August:30-33

    6. Philip GS, Teng SS. Role of respiration in effecting placental transfusion at cesarean section Biol Neonate 1977;31:219-224.

    7. Darwin E. Zoonomia. Vol III 3rd ed London, 1801:302.

    8, Kinmond S, et al. Umbilical cord clamping and preterm infants: a randomized trial, BMJ 1993;306:172175.

    9. Peltonen T. Placental transfusion-advantage and disadvantage. EurJ Pediatr 198 1; 137 141-146,

    10. Saigal S, Usher RH Symptomatic neonatal plethora. Biol Neonate 1977;32:62-72.

    11. Mentzer W. Polycythemia and the hyperviscosity syndrome in newborn infants. Clinics in Haematology 1978;7(1):63-74.

    Answer by Anonymous at 1:12 AM on Jul. 12, 2011

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